How does rhinovirus affect asthma?

Rhinoviruses are a major cause of asthma exacerbations in children and adults. With the use of sensitive RT-PCR methods, respiratory viruses are found in approximately 80% of wheezing episodes in children and in approximately one half of such episodes in adults.

How do you stop airways from remodeling?

The best way to prevent airway remodelling is to control your asthma symptoms. Following a treatment plan and using a controller medication that reduces airway inflammation will result in less remodelling. The less asthma symptoms you experience the less airway remodelling will occur.

What triggers bronchospasm?

Causes of bronchospasm Any swelling or irritation in your airways can cause bronchospasm. This condition commonly affects people with asthma. Other factors that can contribute to bronchospasm include: allergens, such as dust and pet dander.

Is bronchospasm curable?

When that happens, it’s called a bronchial spasm, or a bronchospasm. During a bronchial spasm, breathing becomes more difficult. You may find yourself wheezing as you try to catch your breath. In many cases, bronchial spasms are treatable or preventable.

Can viral infection trigger asthma?

The most common trigger for asthma exacerbation in adults and children, are infection with rhinovirus, though other common respiratory viruses, like parainfluenza virus, cause asthma exacerbation [1–3].

How do you treat rhinovirus?

Rhinovirus (RV) infections are predominantly mild and self-limited; thus, treatment is generally focused on symptomatic relief and prevention of person-to-person spread and complications. The mainstays of therapy include rest, hydration, first-generation antihistamines, and nasal decongestants.

What is Remodelling of the airways?

Airway remodelling refers to the structural changes that occur in both large and small airways relevant to miscellaneous diseases including asthma. In asthma, airway structural changes include subepithelial fibrosis, increased smooth muscle mass, gland enlargement, neovascularization and epithelial alterations.